Церебрастенический синдром представляет собой non-specific neurological syndrome, which is characterized by decreased performance, impaired attention and memory, increased fatigue, as well as headaches, severe depletion of the nervous system and various autonomic manifestations. In addition, there is an increase in the duration of sleep and a decrease in wakefulness, emotional lability of the patient and motor restlessness. Cerebrastenic syndrome in children is often accompanied by an increase in congenital reflexes, a spontaneous Moro reflex and autonomic disorders.
The main causes ofCerebrastenic syndrome are the pathological course of pregnancy and subsequent delivery, which caused a disturbance in the oxygen supply to the baby’s brain. Also, other pathological conditions that trigger the development of this syndrome include medication during pregnancy, intrauterine infections, prematurity, intracranial hemorrhage, sepsis, hemolytic disease, pneumonia.
Until recently, newborns with a diagnosisCerebrastenic syndrome did not survive, but at the present time, thanks to the development of qualified neonatological care, the perinatal mortality rates have dropped significantly. And as a result, the number of children with certain learning difficulties and developmental disorders increased as a long-term result of the pathology of the neonatal period, childbirth and pregnancy. For the same reason, cerebrastenic syndrome occurs in adults.
Cerebrostenic syndromedevelops in children of school and preschool age who have suffered concussion or brain injury, meningitis, encephalitis, as well as common infections with toxic damage to brain tissue. In addition, the disease can develop in most children who have had to undergo surgery, and anemic, circulatory and histotoxic hypoxia are the impetus for its development.
The main clinical manifestations of cerebral syndrome:
- "irritable weakness";
- lethargy, general passivity, sluggishness of movements and thinking;
- increased fatigue and exhaustion, even with minor mental stress;
- low background mood;
- headaches on the background of overwork;
- dizziness and nausea;
- poor tolerance of heat and stuffiness, sudden changes in atmospheric pressure.
These manifestations are characterized by an increase aftervaccination, stressful situations, under the influence of intercurrent diseases and injuries. It is believed that post-traumatic cerebroscence is most characteristic of irritability, irritability, affective explosiveness, as well as hyperesthesia to bright light and sounds, and post-infectious - moodiness, mood swings, tearfulness and feelings of discontent.
As practice shows, the treatment of suchDisorders such as cerebral syndrome should begin immediately after the first clinical manifestations of the disease. Treatment depends on how severely impaired certain functions are expressed, it should be primarily aimed at stimulating and restoring the normal functioning of central regulatory mechanisms. A single generally accepted method of treatment for this disorder does not yet exist. As a rule, therapeutic measures are symptomatic and are based on the experience and intuition of the doctor. So, in the presence of a patient with convulsive syndrome, such drugs as diphenyl, phenobarbital, depakin and finlepsin are prescribed. Amino acids, nootropic drugs and adaptogens are shown to stimulate metabolic processes in brain tissues. Diacarb is used in the treatment of hypertensive syndrome, prozerin in the presence of muscular hypotonia, and dibazol and mydocalm in muscle hypertension syndrome. Cerebral circulation is improved with the help of antispasmodics: Dibazol, aminophylline, pentoxifylline and cinnarizine.
